The Journal of Allergy and Clinical Immunology
Volume 117, Issue 5 , Pages 1008-1013, May 2006

Effect of ciclesonide dose and duration of therapy on exercise-induced bronchoconstriction in patients with asthma

  • Padmaja Subbarao, MD, MSc

      Affiliations

    • From the Hospital for Sick Children, University of Toronto
    • Firestone Institute for Respiratory Health, St Joseph's Hospital, and the Department of Medicine, McMaster University, Hamilton
    • Corresponding Author InformationReprint requests: Padmaja Subbarao, MD, MSc, FRCP(C), Hospital for Sick Children, 555 University Ave, Toronto, Ontario M5G 1X8, Canada.
  • ,
  • Mylinh Duong, MD

      Affiliations

    • Firestone Institute for Respiratory Health, St Joseph's Hospital, and the Department of Medicine, McMaster University, Hamilton
  • ,
  • Ellinor Adelroth, MD, PhD

      Affiliations

    • Umea University, Umea
  • ,
  • Joceline Otis

      Affiliations

    • Firestone Institute for Respiratory Health, St Joseph's Hospital, and the Department of Medicine, McMaster University, Hamilton
  • ,
  • George Obminski, BSc

      Affiliations

    • Firestone Institute for Respiratory Health, St Joseph's Hospital, and the Department of Medicine, McMaster University, Hamilton
  • ,
  • Mark Inman, MD, PhD

      Affiliations

    • Firestone Institute for Respiratory Health, St Joseph's Hospital, and the Department of Medicine, McMaster University, Hamilton
  • ,
  • Soren Pedersen, MD, PhD

      Affiliations

    • University of Southern Denmark, Kolding
  • ,
  • Paul M. O'Byrne, MB

      Affiliations

    • Firestone Institute for Respiratory Health, St Joseph's Hospital, and the Department of Medicine, McMaster University, Hamilton

Toronto and Hamilton, Ontario, Canada, Umea, Sweden, and Kolding, Denmark

Background

Inhaled corticosteroid therapy improves exercise symptoms in asthmatic subjects.

Objective

We sought to evaluate exercise-induced bronchoconstriction (EIB) as a method of determining the dose and time responses of inhaled corticosteroid therapy.

Methods

In this double-blind, randomized, cross-over study with 2 parallel arms, 4 doses of inhaled ciclesonide (40 μg and 160 μg or 80 μg and 320 μg) were compared over 3 weeks of treatment. Twenty-six asthmatic subjects (age range, 14-27 years) with baseline FEV1 values of greater than 70% of predicted value were enrolled. The primary outcome was the maximum percentage decrease in FEV1 after standardized exercise challenge.

Results

After 1 week of therapy, the mean ± SEM reduction in maximum decrease in FEV1 in the ciclesonide 40-μg/80-μg dose group was 9% ± 2.6% (95% CI, 3.9% to 14%), with no additional reduction thereafter. In the ciclesonide 160-μg/320-μg dose group, there was an 8.7% ± 2.5% (95% CI, 3.7% to 13.7%) reduction in maximum decrease in FEV1 after week 1, which continued in a linear fashion during subsequent weeks of treatment. No difference was found between the 2 treatment arms in the temporal response of EIB to ciclesonide treatment. The maximum percentage attenuation in EIB achieved was 51.1% ± 7.9%, which was achieved by using the 320-μg dose after 3 weeks of treatment.

Conclusions

A significant improvement in EIB was demonstrated for all doses of ciclesonide. Use of 160 μg/320 μg of ciclesonide resulted in a continuing improvement in FEV1 with time, and no plateau was seen in protective effect during 3 weeks of treatment.

Clinical implications

Attenuation in exercise-induced decrease can be seen as early as after 1 week of therapy with inhaled ciclesonide at doses greater than 40 μg. However, maximal attenuation in exercise response continues to increase at doses greater than or equal to 200 μg, even after 3 weeks of therapy.

Key words: Asthma, exercise induced, inhaled corticosteroids, randomized controlled trial, dose response

Abbreviations used: AUC, Area under the curve, EIB, Exercise-induced bronchoconstriction, ICS, Inhaled corticosteroid, PEF, Peak expiratory flow

 

 Some of the data from this article have been presented in abstract form at the American Academy of Allergy, Asthma and Immunology Meeting, San Antonio, Tex, March 2005, and the American Thoracic Society Meeting, San Diego, Calif, May 2005.Supported in part by Altana Pharma AG, Germany.Disclosure of potential conflict of interest: S. Pedersen has consultant arrangements with Altana and GlaxoSmithKline and has received grants from AstraZeneca, Altana and GlaxoSmithKline. P. O'Byrne has consultant arrangements with Altana, AstraZeneca, Biolipox, GlaxoSmithKline, Oxygen, Topigen and Roche and has received grants from Altana, AstraZeneca, Biolipox, GlaxoSmithKline, and Pfizer. The rest of the authors have declared that they have no conflict of interest.

PII: S0091-6749(06)00195-3

doi:10.1016/j.jaci.2005.11.048

The Journal of Allergy and Clinical Immunology
Volume 117, Issue 5 , Pages 1008-1013, May 2006